粘附在对照组（PLCL和PGD）上的HUVECs和HUASMCs数量几乎相等。不同的是，PLCL表面两种细胞数量都较少，不利于细胞粘附。而PGD材料表面上ECs和SMCs的粘附数量明显较多，但HUVECs/HUASMCs的粘附比率仍没有明显增加（仅为1.0 ± 0.1）。重要的是，在孵育4小时后，所有CAG接枝的表面（PGD-CAG和PGD-CAG/ACH₁₁），其HUVECs的数量有所增加，而HUASMCs的数量明显减少。这主要归因于CAG多肽对ECs/SMCs粘附能力的影响不同。此外，PGD-CAG/ACH₁₁表面的HUVECs/HUASMCs的粘附比率（2.2 ± 0.3）显著高于PGD（1.0 ± 0.2）或PGD-ACH₁₁组（0.9 ± 0.1）。这有力证明了PGD-CAG/ACH₁₁纤维表面的ECs选择性粘附， 故在实现血管的原位快速内皮化方面具有巨大潜力。

The results showed that the amount of HUVECs and HUASMCs adherent on the controls (PLCL and PGD) was almost equal, which resist both ECs and SMCs. As expected, the surface functionalizing with PDA promoted both ECs and SMCs adhesion as compared to the controls, but there was no significant increase in the adhesion ratio of HUVECs/HUASMCs (only 1.0 ± 0.1). However, after 4 h incubation on all CAG-coating surfaces (PGD-CAG and PGD-CAG/ACH₁₁), an increase in the amount of HUVECs and a remarkable decrease in HUASMCs were observed, mainly attributed to the biological effect on ECs/SMCs adhesion of CAG peptide. Importantly, a significantly increased ratio of HUVECs/HUASMCs (2.2 ± 0.3) was observed on the PGD-CAG/ACH₁₁ surfaces compared with PGD (1.0 ± 0.2) or PGD-ACH₁₁ group (0.9 ± 0.1). The result of HUVECs/HUASMCs competitive adhesion was also verified the ECs-selectivity of multifunctional coating. All above results strongly suggested that PGD-CAG/ACH₁₁ coating benefited for HUVECs growth behavior and avoided the excessive proliferation of SMCs, indicating a great potential for accelerating the vascular in situ endothelialization.